Module 5 Abnormal Gait 3 B Pathological Differences
Module 5 Abnormal Gait 3 B: Pathological Differences: Foot and Ankle Deviations
References § Perry, J and Burnfield, J. (2010). Gait Analysis: Normal and Pathological Function 2 nd edition. Ch 9. § Hsu, JD; Michael, JW and Fisk, JR. (2008) Atlas of Orthoses and Assistive Devices, 4 th edition. Ch 5, pgs 67 to 74.
Agenda - Pathological Gait: Ankle and Foot Variations • 1. Floor Contact Variations • • • A. B. C. D. E. Forefoot Contact Delayed Heel Contact Foot Flat Contact Low Heel Foot Slap • 2. Ankle Variations • • A. B. C. D. Excess Plantarflexion Excess Dorsiflexion Prolonged Heel Only Premature Heel-Off • 3. Subtalar Joint Variations • A. Excess Inversion • B. Excess Eversion • 4. Toe Variations • A. Excess Toe Extension • B. Limited Toe Extension • C. Clawed Toes
1. Floor Contact Variations • A. Forefoot Contact • Disrupts: heel rocker, forward progression of tibia and shock absorption of knee • Caused by: • Three common resulting loading patterns:
1. Floor Contact Variations • B. Delayed Heel Contact • Disrupts: heel rocker and forward progression • Commonly caused by yielding elastic PF contracture or spasticity • Therefore assessment of PF tightness vs. contracture is imperative • Delayed Heel Contact Video
1. Floor Contact Deviations • C. Foot Flat Contact • Disrupts: heel rocker and forward progression • Caused by: • Video of Pediatric Foot Flat Contact Left Limb
1. Floor Contact Variations, continued • D. Low Heel • Disrupts: heel rocker and forward progression • Caused by: • Very difficult to observe clinically
1. Floor Contact Variations Foot Slap • E. Foot Slap • Disrupts: heel rocker, forward progression and shock absorption • Caused by:
2. Ankle Variations • A. Excessive Plantar Flexion = “inadequate DF” • Disrupts: rockers during stance and toe clearance and limb advancement during swing • Focus now on just Mid Stance and Mid Swing
Pathological Gait • Variations observed during Stance • Variations observed during Swing • Observations made coronally and sagittally
2. Ankle Variations Excessive Dorsiflexion • B. Excessive Dorsiflexion = “inadequate PF” • Disrupts: controlled forward progression of tibia • Early Stance and Later Stance: • Accentuates heel rocker – causes more demand on quads • During pre-swing, shortens limb – causes reduced pelvis support • Limits toe rocker
2. Ankle Variations Heel • C. Prolonged Heel Only • Infrequent finding • Disrupts: stability and forward progression • D. Premature Heel-Off • Disrupts: heel and ankle rockers and associated forward progression • Difficult to see in later part of Stance
2. Ankle Variations • E. No Heel Off/Delayed Heel Off • • Disrupts forefoot and toe rockers Reduces forward progression Shortens contralateral step length Limits Pre-Swing knee flexion
2. Ankle Variations, continued • F. Drag • Disrupts: forward progression and foot clearance • G. Contralateral Vaulting • Decreases contralateral stance stability and increases calf muscle demand • Initial, mid- and terminal Swing
3. Subtalar Variations • A. Excess Inversion = excessive lateral foot contact • A. k. a. “varus” • Swing = difficulty with clearance • Stance = rigid foot less able to absorb shock
3. Subtalar Variations Excess Eversion • B. Excess Eversion = excessive medial foot contact • A. k. a. “valgus” • WA = increases rotary stress on ankle and knee • SLS = prevents achieving rigid forefoot forward progression • Usually result of total weakness of invertors vs. excessive peroneals
4. Toe Variations • A. Excess Toe Extension • During WA = may reflect increased use of long toe extensors if Tibialis Anterior is weak • During SLA, may assist with foot clearance • B. Limited Toe Extension • Disrupts: forefoot and toe rockers • Reduces contra-lateral step length
4. Toe Variations • C. Clawed Toes • Disrupts: forefoot and toe rockers • Reduces contra-lateral step length
So, what do you see? Focus on Ankle and Foot Variations
Foot/Ankle Complex Sagittal Plane Motion
The End
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