Acute inflammation Formation of acute inflammatory exudate From

  • Slides: 27
Download presentation
Acute inflammation

Acute inflammation

Formation of acute inflammatory exudate From: Stevens A. J Lowe J. Pathology. Mosby 1995

Formation of acute inflammatory exudate From: Stevens A. J Lowe J. Pathology. Mosby 1995 Chemical mediators acting on nearby blood vessels from inflammatory focus Fig. 5. 1.

Vasodilation and increased permiability Fig. 5. 2.

Vasodilation and increased permiability Fig. 5. 2.

Neutrophils margination and emigration Fig. 5. 3.

Neutrophils margination and emigration Fig. 5. 3.

Neutrophil margination, emigration and emigration in acute inflammation From: Stevens A. J Lowe J.

Neutrophil margination, emigration and emigration in acute inflammation From: Stevens A. J Lowe J. Pathology. Mosby 1995 Neutrophil margination, emigration and emigration in acute inflammation Fig. 5. 4.

Neutrophil margination and emigration in acute inflammation Fig. 5. 5.

Neutrophil margination and emigration in acute inflammation Fig. 5. 5.

Neutrophil margination, emigration and emigration in acute inflammation Fig. 5. 6.

Neutrophil margination, emigration and emigration in acute inflammation Fig. 5. 6.

Neutropil phagocytosis From: Stevens A. J Lowe J. Pathology. Mosby 1995 Fig. 5. 7.

Neutropil phagocytosis From: Stevens A. J Lowe J. Pathology. Mosby 1995 Fig. 5. 7.

Mechanisms involved in inflammation-From: Stevens A. J Lowe J. Pathology. Mosby 1995 Fig. 5.

Mechanisms involved in inflammation-From: Stevens A. J Lowe J. Pathology. Mosby 1995 Fig. 5. 8.

Acute serous inflammation From: Stevens A. J Lowe J. Pathology. Mosby 1995 Acute amigdalitis

Acute serous inflammation From: Stevens A. J Lowe J. Pathology. Mosby 1995 Acute amigdalitis Fig. 5. 9.

Herpetic inflammation Fig. 5. 10.

Herpetic inflammation Fig. 5. 10.

Serous alveolitis From cases of the Pathology Department U. M. F. “Gr. T. Popa”

Serous alveolitis From cases of the Pathology Department U. M. F. “Gr. T. Popa” Iasi Fig. 5. 11.

Fig. 5. 12. Serous alveolitis: (1) Alveolar walls (congestion of parieto-alveolar capillaries); (2) Alveolar

Fig. 5. 12. Serous alveolitis: (1) Alveolar walls (congestion of parieto-alveolar capillaries); (2) Alveolar lumen: serous exudate.

Fibrinous inflammation From cases of the Pathology Department U. M. F. “Gr. T. Popa”

Fibrinous inflammation From cases of the Pathology Department U. M. F. “Gr. T. Popa” Iasi Mallory staining Fig. 5. 13.

Mallory staining Fibrinous alveolitis: (1) alveolar walls (congestion of parieto-alveolar capillaries); (2) alveolar lumen:

Mallory staining Fibrinous alveolitis: (1) alveolar walls (congestion of parieto-alveolar capillaries); (2) alveolar lumen: fibrinous exudate. Fig. 5. 14.

Fibrinous pericarditis From: Stevens A. J Lowe J. Pathology. Mosby 1995 Fig. 5. 15.

Fibrinous pericarditis From: Stevens A. J Lowe J. Pathology. Mosby 1995 Fig. 5. 15. Fibrinous deposits on parietal pericardium

Fibrinous pericarditis: (1) Epicardium is covered by fibrinous exudate forming intense eosinophilic fibrin network;

Fibrinous pericarditis: (1) Epicardium is covered by fibrinous exudate forming intense eosinophilic fibrin network; (2) Epicardium with vascular congestion and neutrophils. From cases of the Pathology Department U. M. F. “Gr. T. Popa” Iasi Fig. 5. 16.

Acute purulent inflammation Fig. 5. 17. Pus smear (May Grunwald Giemsa) Purulent exudate (pus)

Acute purulent inflammation Fig. 5. 17. Pus smear (May Grunwald Giemsa) Purulent exudate (pus) is composed of: neutrophiles; macrophages; eritrocytes; necrotic debris; fibrin; bacteria.

Fig. 5. 18. Pus smear (MGG) From cases of the Pathology Department - U.

Fig. 5. 18. Pus smear (MGG) From cases of the Pathology Department - U. M. F. “Gr. T. Popa” Iasi

Acute diffuse purulent inflammation From: Stevens A. J Lowe J. Pathology. Mosby 1995 Fig.

Acute diffuse purulent inflammation From: Stevens A. J Lowe J. Pathology. Mosby 1995 Fig. 5. 19. Purulent leptomeningitis

Fig. 5. 20. HE staining Purulent leptomeningitis: (1) meninges is diffusely thickened by purulent

Fig. 5. 20. HE staining Purulent leptomeningitis: (1) meninges is diffusely thickened by purulent exudate and vascular congestion. (2) - congested vessels (leukocyte margination and diapedesis). From cases of the Pathology Department - U. M. F. “Gr. T. Popa” Iasi

HE staining Vascular congestion and leukocyte diapedesis Fig. 5. 21.

HE staining Vascular congestion and leukocyte diapedesis Fig. 5. 21.

HE staining Fig. 5. 22. Leukocyte margination and diapedesis From cases of the Pathology

HE staining Fig. 5. 22. Leukocyte margination and diapedesis From cases of the Pathology Department - U. M. F. “Gr. T. Popa

Acute localised purulent inflammation Lung acute and chronic abscesses From: Stevens A. J Lowe

Acute localised purulent inflammation Lung acute and chronic abscesses From: Stevens A. J Lowe J. Pathology. Mosby 1995 Fig. 5. 23.

Pulmonary abscess From: Stevens A. J Lowe J. Pathology. Mosby 1995 Fig. 5. 24.

Pulmonary abscess From: Stevens A. J Lowe J. Pathology. Mosby 1995 Fig. 5. 24. Pulmonary abscess: (1) the cavity: contains a suppurative material and air content (in case of communication with air conducts); (2) wall: (a) acute abscess – the wall has irregular borders reprezented by suppurative necrotic lung parenchyma; (b) chronic abscess - the wall is a pyogenic membrane that becomes fibrotic by connective organization.

Localised purulent inflammation (abscess) • • Abscess: – Localized purulent Inflammation; Types – acute

Localised purulent inflammation (abscess) • • Abscess: – Localized purulent Inflammation; Types – acute / recent – chronic Recent cerebral abscess • center: – purulent exudate; • periphery: – fibrin wall. Fig. 5. 25. From cases of the Pathology Department U. M. F. “Gr. T. Popa” Iasi HE staining

Cronic hepatic abscess • central cavity contains • purulent exudate; § peripheral wall -

Cronic hepatic abscess • central cavity contains • purulent exudate; § peripheral wall - pyogenic membrane § external layer § connective-vascular tissue of neoformation § internal layer § fibrin network § neutrophils Fig. 5. 26. From cases of the Pathology Department U. M. F. “Gr. T. Popa” Iasi

ACUTE INFLAMMATION ACUTE INFLAMMATION COMPONENT Definition Acute inflammationACUTE INFLAMMATION ACUTE INFLAMMATION COMPONENT Definition Acute inflammation
Inflammation Acute inflammation The cardinal signs of inflammationInflammation Acute inflammation The cardinal signs of inflammation
Acute Inflammation Dr Shoaib Raza Acute Inflammation ResponseAcute Inflammation Dr Shoaib Raza Acute Inflammation Response
Inflammatory Process Inflammation What is Inflammation A vascularInflammatory Process Inflammation What is Inflammation A vascular
UNIT I CELL OF INFLAMMATION INFLAMMATION THE INFLAMMATORYUNIT I CELL OF INFLAMMATION INFLAMMATION THE INFLAMMATORY
CHRONIC INFLAMMATION CHRONIC INFLAMMATION Prolonged inflammatory response toCHRONIC INFLAMMATION CHRONIC INFLAMMATION Prolonged inflammatory response to
INFLAMMATION Comprehensive Approach Inflammation and repair Inflammation isINFLAMMATION Comprehensive Approach Inflammation and repair Inflammation is
Inflammation VLA October 4 2011 Inflammation n InflammationInflammation VLA October 4 2011 Inflammation n Inflammation
Inflammation 14 11 2004 Inflammation Inflammation is theInflammation 14 11 2004 Inflammation Inflammation is the
UNIT I Inflammation INFLAMMATION CHRONIC INFLAMMATION DEFINITION ANDUNIT I Inflammation INFLAMMATION CHRONIC INFLAMMATION DEFINITION AND
Inflammation Inflammation Definition Inflammation is biochemical structural andInflammation Inflammation Definition Inflammation is biochemical structural and
UNIT I INFLAMMATION INFLAMMATION INFLAMMATION Local response ofUNIT I INFLAMMATION INFLAMMATION INFLAMMATION Local response of
Inflammation JongMo Seo Inflammation JongMo Seo Inflammation JongMoInflammation JongMo Seo Inflammation JongMo Seo Inflammation JongMo
Acute Inflammatory Dermatoses Literally thousands of inflammatory dermatosesAcute Inflammatory Dermatoses Literally thousands of inflammatory dermatoses
Acute and Chronic inflammation Fatima Obeidat MD InflammationAcute and Chronic inflammation Fatima Obeidat MD Inflammation
MORPHOLOGICAL PATTERNS OF INFLAMMATION 1 PATTERNS ACUTE INFLAMMATIONMORPHOLOGICAL PATTERNS OF INFLAMMATION 1 PATTERNS ACUTE INFLAMMATION
PRACTICAL 2 Inflammation I Acute Inflammation Pathology DeptPRACTICAL 2 Inflammation I Acute Inflammation Pathology Dept
Acute and chronic inflammation Inflammation what is itAcute and chronic inflammation Inflammation what is it
Chronic inflammation Outcomes of acute inflammation progression toChronic inflammation Outcomes of acute inflammation progression to
PRACTICAL Inflammation I Acute Inflammation Foundation Block 2015PRACTICAL Inflammation I Acute Inflammation Foundation Block 2015
Acute Inflammation Introduction Inflammation is the response ofAcute Inflammation Introduction Inflammation is the response of
Types of acute inflammation I Suppurative inflammation associatedTypes of acute inflammation I Suppurative inflammation associated
ACUTE INFLAMMATORY DESEASES OF LUNGS PNEUMONIAS ACUTE DESTRUCTIVEACUTE INFLAMMATORY DESEASES OF LUNGS PNEUMONIAS ACUTE DESTRUCTIVE
Acute otitis media Acute inflammation in middle earAcute otitis media Acute inflammation in middle ear